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Artigo de periodico
Human herpesvirus 8 in perinatally HIV-infected children with interstitial lung disease (2018)
Scotta, Marcelo Comerlato; Fabro, Alexandre Todorovic; Machado, Paula Renata Lima; Ramos, Simone Gusmão; Cervi, Maria Célia; Fonseca, Benedito Antônio Lopes da; Motta, Fabrizio; Negrini, Bento Vidal de Moura
Source: Journal of Tropical Pediatrics. Unidade: FMRP
Subjects: HIV, CRIANÇAS, DOENÇA PULMONAR (ESPECIALIDADE)
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ABNT
SCOTTA, Marcelo Comerlato et al. Human herpesvirus 8 in perinatally HIV-infected children with interstitial lung disease. Journal of Tropical Pediatrics, v. 64, n. 5, p. 382-388, 2018Tradução . . Disponível em: https://doi.org/10.1093/tropej/fmx080. Acesso em: 28 abr. 2024.
APA
Scotta, M. C., Fabro, A. T., Machado, P. R. L., Ramos, S. G., Cervi, M. C., Fonseca, B. A. L. da, et al. (2018). Human herpesvirus 8 in perinatally HIV-infected children with interstitial lung disease. Journal of Tropical Pediatrics, 64( 5), 382-388. doi:10.1093/tropej/fmx080
NLM
Scotta MC, Fabro AT, Machado PRL, Ramos SG, Cervi MC, Fonseca BAL da, Motta F, Negrini BV de M. Human herpesvirus 8 in perinatally HIV-infected children with interstitial lung disease [Internet]. Journal of Tropical Pediatrics. 2018 ; 64( 5): 382-388.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1093/tropej/fmx080
Vancouver
Scotta MC, Fabro AT, Machado PRL, Ramos SG, Cervi MC, Fonseca BAL da, Motta F, Negrini BV de M. Human herpesvirus 8 in perinatally HIV-infected children with interstitial lung disease [Internet]. Journal of Tropical Pediatrics. 2018 ; 64( 5): 382-388.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1093/tropej/fmx080
Artigo de periodico
Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils (2018)
Cavalcanti Neto, Marinaldo Pacífico; Prado, Rafael de Queiroz; Piñeros, Annie Rocio; Sérgio, Cássia Alves; Bertolini, Thaís Barboza ; Gembre, Ana Flávia; Ramos, Simone Gusmão ; Bonato, Vânia Luiza Deperon
Source: Tuberculosis. Unidade: FMRP
Subjects: TUBERCULOSE, SEQUENCIAMENTO GENÉTICO, TESTES DE SENSIBILIDADE MICROBIANA, MUTAÇÃO, MYCOBACTERIUM TUBERCULOSIS
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ABNT
CAVALCANTI NETO, Marinaldo Pacífico et al. Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils. Tuberculosis, v. 113, p. 1-9, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.tube.2018.08.009. Acesso em: 28 abr. 2024.
APA
Cavalcanti Neto, M. P., Prado, R. de Q., Piñeros, A. R., Sérgio, C. A., Bertolini, T. B., Gembre, A. F., et al. (2018). Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils. Tuberculosis, 113, 1-9. doi:10.1016/j.tube.2018.08.009
NLM
Cavalcanti Neto MP, Prado R de Q, Piñeros AR, Sérgio CA, Bertolini TB, Gembre AF, Ramos SG, Bonato VLD. Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils [Internet]. Tuberculosis. 2018 ; 113 1-9.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.tube.2018.08.009
Vancouver
Cavalcanti Neto MP, Prado R de Q, Piñeros AR, Sérgio CA, Bertolini TB, Gembre AF, Ramos SG, Bonato VLD. Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils [Internet]. Tuberculosis. 2018 ; 113 1-9.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.tube.2018.08.009
Artigo de periodico
Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model (2018)
Martins, Larissa M. S.; Perez, Malena M.; Pereira, Camila A.; Costa, Frederico R. C.; Dias, Murilo S.; Tostes, Rita de Cássia Aleixo; Ramos, Simone Gusmão; Zoete, Marcel R. de; Ryffel, Bernhard; Silva, João Santana da; Carlos, Daniela
Source: Immunology. Unidade: FMRP
Subjects: INFLAMAÇÃO, LINFÓCITOS, INTERLEUCINAS, DOENÇAS METABÓLICAS, OBESIDADE
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ABNT
MARTINS, Larissa M. S. et al. Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model. Immunology, v. 154, n. 4, p. 624-636, 2018Tradução . . Disponível em: https://doi.org/10.1111/imm.12946. Acesso em: 28 abr. 2024.
APA
Martins, L. M. S., Perez, M. M., Pereira, C. A., Costa, F. R. C., Dias, M. S., Tostes, R. de C. A., et al. (2018). Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model. Immunology, 154( 4), 624-636. doi:10.1111/imm.12946
NLM
Martins LMS, Perez MM, Pereira CA, Costa FRC, Dias MS, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Carlos D. Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model [Internet]. Immunology. 2018 ; 154( 4): 624-636.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1111/imm.12946
Vancouver
Martins LMS, Perez MM, Pereira CA, Costa FRC, Dias MS, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Carlos D. Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model [Internet]. Immunology. 2018 ; 154( 4): 624-636.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1111/imm.12946
Artigo de periodico
Doxycycline balance the TIMPs 1 and 2 expressions in a new model of abdominal aortic aneurysms (2018)
Mata, Karina Magalhães; Fernandes, Cleverson Rodrigues ; Tefé-Silva, Cristiane; Ramos, Simone Gusmão
Source: Journal of Immunological Sciences. Unidade: FMRP
Subjects: ANEURISMA AÓRTICO, ABDOME, ANTIBIÓTICOS
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ABNT
MATA, Karina Magalhães et al. Doxycycline balance the TIMPs 1 and 2 expressions in a new model of abdominal aortic aneurysms. Journal of Immunological Sciences, v. 2, n. 6, p. 4-8, 2018Tradução . . Acesso em: 28 abr. 2024.
APA
Mata, K. M., Fernandes, C. R., Tefé-Silva, C., & Ramos, S. G. (2018). Doxycycline balance the TIMPs 1 and 2 expressions in a new model of abdominal aortic aneurysms. Journal of Immunological Sciences, 2( 6), 4-8.
NLM
Mata KM, Fernandes CR, Tefé-Silva C, Ramos SG. Doxycycline balance the TIMPs 1 and 2 expressions in a new model of abdominal aortic aneurysms. Journal of Immunological Sciences. 2018 ; 2( 6): 4-8.[citado 2024 abr. 28 ]
Vancouver
Mata KM, Fernandes CR, Tefé-Silva C, Ramos SG. Doxycycline balance the TIMPs 1 and 2 expressions in a new model of abdominal aortic aneurysms. Journal of Immunological Sciences. 2018 ; 2( 6): 4-8.[citado 2024 abr. 28 ]
Artigo de periodico
Cytotoxicity of peracetic acid: evaluation of effects on metabolism, structure and cell death (2018)
Viola, K. S.; Rodrigues, E. M.; Tanomaru-Filho, M.; Carlos, I. Z.; Ramos, Simone Gusmão; Guerreiro-Tanomaru, J. M.; Faria, G.
Source: International Endodontic Journal. Unidade: FMRP
Subjects: CITOTOXICIDADE IMUNOLÓGICA, ENDODONTIA, FIBROBLASTOS, HIPOCLORITO DE SÓDIO
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ABNT
VIOLA, K. S. et al. Cytotoxicity of peracetic acid: evaluation of effects on metabolism, structure and cell death. International Endodontic Journal, v. 51, p. e264-e277, 2018Tradução . . Disponível em: https://doi.org/10.1111/iej.12750. Acesso em: 28 abr. 2024.
APA
Viola, K. S., Rodrigues, E. M., Tanomaru-Filho, M., Carlos, I. Z., Ramos, S. G., Guerreiro-Tanomaru, J. M., & Faria, G. (2018). Cytotoxicity of peracetic acid: evaluation of effects on metabolism, structure and cell death. International Endodontic Journal, 51, e264-e277. doi:10.1111/iej.12750
NLM
Viola KS, Rodrigues EM, Tanomaru-Filho M, Carlos IZ, Ramos SG, Guerreiro-Tanomaru JM, Faria G. Cytotoxicity of peracetic acid: evaluation of effects on metabolism, structure and cell death [Internet]. International Endodontic Journal. 2018 ; 51 e264-e277.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1111/iej.12750
Vancouver
Viola KS, Rodrigues EM, Tanomaru-Filho M, Carlos IZ, Ramos SG, Guerreiro-Tanomaru JM, Faria G. Cytotoxicity of peracetic acid: evaluation of effects on metabolism, structure and cell death [Internet]. International Endodontic Journal. 2018 ; 51 e264-e277.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1111/iej.12750
Artigo de periodico
High-fat and fat-enriched diets impair the benefits of moderate physical training in the aorta and the heart in rats (2017)
Fernandes, Cleverson Rodrigues; Kannen, Vinicius; Mata, Karina Magalhães; Frajacomo, Fernando Tadeu; Jordão Junior, Alceu Afonso; Gasparotto, Bianca; Sakita, Juliana Yumi; Elias Junior, Jorge; Leonardi, Daphne Santoro; Mauad, Fernando Marum; Ramos, Simone Gusmão; Uyemura, Sergio Akira; Garcia, Sergio Britto
Source: Frontiers in Nutrition. Unidades: FMRP, FCFRP
Subjects: BIOQUÍMICA, VASOS SANGUÍNEOS, CORAÇÃO, OBESIDADE, NATAÇÃO
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ABNT
FERNANDES, Cleverson Rodrigues et al. High-fat and fat-enriched diets impair the benefits of moderate physical training in the aorta and the heart in rats. Frontiers in Nutrition, v. 4, 2017Tradução . . Disponível em: https://doi.org/10.3389/fnut.2017.00021. Acesso em: 28 abr. 2024.
APA
Fernandes, C. R., Kannen, V., Mata, K. M., Frajacomo, F. T., Jordão Junior, A. A., Gasparotto, B., et al. (2017). High-fat and fat-enriched diets impair the benefits of moderate physical training in the aorta and the heart in rats. Frontiers in Nutrition, 4. doi:10.3389/fnut.2017.00021
NLM
Fernandes CR, Kannen V, Mata KM, Frajacomo FT, Jordão Junior AA, Gasparotto B, Sakita JY, Elias Junior J, Leonardi DS, Mauad FM, Ramos SG, Uyemura SA, Garcia SB. High-fat and fat-enriched diets impair the benefits of moderate physical training in the aorta and the heart in rats [Internet]. Frontiers in Nutrition. 2017 ; 4[citado 2024 abr. 28 ] Available from: https://doi.org/10.3389/fnut.2017.00021
Vancouver
Fernandes CR, Kannen V, Mata KM, Frajacomo FT, Jordão Junior AA, Gasparotto B, Sakita JY, Elias Junior J, Leonardi DS, Mauad FM, Ramos SG, Uyemura SA, Garcia SB. High-fat and fat-enriched diets impair the benefits of moderate physical training in the aorta and the heart in rats [Internet]. Frontiers in Nutrition. 2017 ; 4[citado 2024 abr. 28 ] Available from: https://doi.org/10.3389/fnut.2017.00021
Artigo de periodico
Leukotriene B4 is essential for lung host defence and alpha-defensin-1 production during Achromobacter xylosoxidans infection (2017)
Prado, Morgana K. B.; Locachevic, Gisele A.; Zoccal, Karina F.; Paula-Silva, Francisco Wanderley Garcia de; Fontanari, Caroline; Ferreira, Joseane Cristina; Pereira, Priscilla A. T.; Gardinassi, Luiz G.; Ramos, Simone Gusmão; Sorgi, Carlos Arterio; Darini, Ana Lúcia da Costa; Faccioli, Lucia Helena
Source: Scientific Reports. Unidades: FORP, FCFRP, FMRP
Subjects: LEUCOTRIENOS B, PSEUDOMONAS, INFECÇÕES BACTERIANAS GRAM-NEGATIVAS
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ABNT
PRADO, Morgana K. B. et al. Leukotriene B4 is essential for lung host defence and alpha-defensin-1 production during Achromobacter xylosoxidans infection. Scientific Reports, v. 7, n. 1, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-17993-9. Acesso em: 28 abr. 2024.
APA
Prado, M. K. B., Locachevic, G. A., Zoccal, K. F., Paula-Silva, F. W. G. de, Fontanari, C., Ferreira, J. C., et al. (2017). Leukotriene B4 is essential for lung host defence and alpha-defensin-1 production during Achromobacter xylosoxidans infection. Scientific Reports, 7( 1). doi:10.1038/s41598-017-17993-9
NLM
Prado MKB, Locachevic GA, Zoccal KF, Paula-Silva FWG de, Fontanari C, Ferreira JC, Pereira PAT, Gardinassi LG, Ramos SG, Sorgi CA, Darini AL da C, Faccioli LH. Leukotriene B4 is essential for lung host defence and alpha-defensin-1 production during Achromobacter xylosoxidans infection [Internet]. Scientific Reports. 2017 ; 7( 1):[citado 2024 abr. 28 ] Available from: https://doi.org/10.1038/s41598-017-17993-9
Vancouver
Prado MKB, Locachevic GA, Zoccal KF, Paula-Silva FWG de, Fontanari C, Ferreira JC, Pereira PAT, Gardinassi LG, Ramos SG, Sorgi CA, Darini AL da C, Faccioli LH. Leukotriene B4 is essential for lung host defence and alpha-defensin-1 production during Achromobacter xylosoxidans infection [Internet]. Scientific Reports. 2017 ; 7( 1):[citado 2024 abr. 28 ] Available from: https://doi.org/10.1038/s41598-017-17993-9
Artigo de periodico
Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure (2017)
Prado, Fernanda Peres; Santos, Daniele Oliveira dos; Blefari, Valdecir; Silva, Carlos Alberto Aguiar da; Machado, Juliano; Kettelhut, Isis do Carmo; Ramos, Simone Gusmão; Baruffi, Marcelo Dias; Salgado, Hélio César; Prado, Cibele Maria
Source: PLOS ONE. Unidades: FMRP, FCFRP, FZEA
Subjects: BIOMARCADORES, PRESSÃO SANGUÍNEA, CARDIOMEGALIA, DIAGNÓSTICO POR IMAGEM, ECOCARDIOGRAFIA
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ABNT
PRADO, Fernanda Peres et al. Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure. PLOS ONE, v. 12, n. 12, 2017Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0189469. Acesso em: 28 abr. 2024.
APA
Prado, F. P., Santos, D. O. dos, Blefari, V., Silva, C. A. A. da, Machado, J., Kettelhut, I. do C., et al. (2017). Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure. PLOS ONE, 12( 12). doi:10.1371/journal.pone.0189469
NLM
Prado FP, Santos DO dos, Blefari V, Silva CAA da, Machado J, Kettelhut I do C, Ramos SG, Baruffi MD, Salgado HC, Prado CM. Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure [Internet]. PLOS ONE. 2017 ; 12( 12):[citado 2024 abr. 28 ] Available from: https://doi.org/10.1371/journal.pone.0189469
Vancouver
Prado FP, Santos DO dos, Blefari V, Silva CAA da, Machado J, Kettelhut I do C, Ramos SG, Baruffi MD, Salgado HC, Prado CM. Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure [Internet]. PLOS ONE. 2017 ; 12( 12):[citado 2024 abr. 28 ] Available from: https://doi.org/10.1371/journal.pone.0189469
Artigo de periodico
Increased atrial β-adrenergic receptors and GRK-2 gene expression can play a fundamental role in heart failure after repair of congenital heart disease with cardiopulmonary bypass (2017)
Oliveira, Marcela Silva; Carmona, Fabio; Vicente, Walter Villela de Andrade; Manso, Paulo Henrique; Mata, Karina Magalhães Alves da; Celes, Mara Rúbia Nunes; Campos, Erica C.; Ramos, Simone Gusmão
Source: Pediatric Cardiology. Unidade: FMRP
Subjects: EXPRESSÃO GÊNICA, BIÓPSIA, INSUFICIÊNCIA CARDÍACA, BEBÊS, MIOCÁRDIO, BIOMARCADORES
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ABNT
OLIVEIRA, Marcela Silva et al. Increased atrial β-adrenergic receptors and GRK-2 gene expression can play a fundamental role in heart failure after repair of congenital heart disease with cardiopulmonary bypass. Pediatric Cardiology, v. 38, n. 4, p. 734-745, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00246-017-1573-1. Acesso em: 28 abr. 2024.
APA
Oliveira, M. S., Carmona, F., Vicente, W. V. de A., Manso, P. H., Mata, K. M. A. da, Celes, M. R. N., et al. (2017). Increased atrial β-adrenergic receptors and GRK-2 gene expression can play a fundamental role in heart failure after repair of congenital heart disease with cardiopulmonary bypass. Pediatric Cardiology, 38( 4), 734-745. doi:10.1007/s00246-017-1573-1
NLM
Oliveira MS, Carmona F, Vicente WV de A, Manso PH, Mata KMA da, Celes MRN, Campos EC, Ramos SG. Increased atrial β-adrenergic receptors and GRK-2 gene expression can play a fundamental role in heart failure after repair of congenital heart disease with cardiopulmonary bypass [Internet]. Pediatric Cardiology. 2017 ; 38( 4): 734-745.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1007/s00246-017-1573-1
Vancouver
Oliveira MS, Carmona F, Vicente WV de A, Manso PH, Mata KMA da, Celes MRN, Campos EC, Ramos SG. Increased atrial β-adrenergic receptors and GRK-2 gene expression can play a fundamental role in heart failure after repair of congenital heart disease with cardiopulmonary bypass [Internet]. Pediatric Cardiology. 2017 ; 38( 4): 734-745.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1007/s00246-017-1573-1
Artigo de periodico
M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection (2017)
Piñeros, A. R.; Campos, L. W.; Fonseca, Denise Morais da ; Bertolini, T. B.; Gembre, A. F.; Prado, R. Q.; Alves Filho, José Carlos Farias; Ramos, Simone Gusmão; Russo, Momtchilo; Bonato, Vânia Luiza Deperon
Source: Scientific Reports. Unidades: ICB, FMRP
Subjects: IMUNOLOGIA, ALÉRGENOS, INTERLEUCINAS, MACRÓFAGOS, MYCOBACTERIUM TUBERCULOSIS, TUBERCULOSE PULMONAR
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ABNT
PIÑEROS, A. R. et al. M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection. Scientific Reports, v. 7, n. 41240, p. 1-12, 2017Tradução . . Disponível em: https://doi.org/10.1038/srep41240. Acesso em: 28 abr. 2024.
APA
Piñeros, A. R., Campos, L. W., Fonseca, D. M. da, Bertolini, T. B., Gembre, A. F., Prado, R. Q., et al. (2017). M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection. Scientific Reports, 7( 41240), 1-12. doi:10.1038/srep41240
NLM
Piñeros AR, Campos LW, Fonseca DM da, Bertolini TB, Gembre AF, Prado RQ, Alves Filho JCF, Ramos SG, Russo M, Bonato VLD. M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection [Internet]. Scientific Reports. 2017 ; 7( 41240): 1-12.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1038/srep41240
Vancouver
Piñeros AR, Campos LW, Fonseca DM da, Bertolini TB, Gembre AF, Prado RQ, Alves Filho JCF, Ramos SG, Russo M, Bonato VLD. M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection [Internet]. Scientific Reports. 2017 ; 7( 41240): 1-12.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1038/srep41240
Artigo de periodico
A dark bronchoalveolar lavage fluid (2017)
Martinez-Baddini, José; Alves, Luis Renato; Ramos, Simone Gusmão
Source: The American Journal of the Medical Sciences. Unidade: FMRP
Subjects: BRONQUIOLITE, DOENÇAS PULMONARES INTERSTICIAIS
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ABNT
MARTINEZ-BADDINI, José e ALVES, Luis Renato e RAMOS, Simone Gusmão. A dark bronchoalveolar lavage fluid. The American Journal of the Medical Sciences, v. 353, n. 4, p. 413, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.amjms.2016.06.010. Acesso em: 28 abr. 2024.
APA
Martinez-Baddini, J., Alves, L. R., & Ramos, S. G. (2017). A dark bronchoalveolar lavage fluid. The American Journal of the Medical Sciences, 353( 4), 413. doi:10.1016/j.amjms.2016.06.010
NLM
Martinez-Baddini J, Alves LR, Ramos SG. A dark bronchoalveolar lavage fluid [Internet]. The American Journal of the Medical Sciences. 2017 ; 353( 4): 413.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.amjms.2016.06.010
Vancouver
Martinez-Baddini J, Alves LR, Ramos SG. A dark bronchoalveolar lavage fluid [Internet]. The American Journal of the Medical Sciences. 2017 ; 353( 4): 413.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.amjms.2016.06.010
Artigo de periodico
Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model (2017)
Carlos, Daniela ; Costa, Frederico Ribeiro Campos ; Pereira, Camila André; Rocha, Fernanda Agostini; Yaochite, Juliana Navarro Ueda; Oliveira, Gabriela G.; Carneiro, Fernando Silva ; Tostes, Rita de Cássia Aleixo ; Ramos, Simone Gusmão ; Zamboni, Dario Simões ; Câmara, Niels Olsen Saraiva ; Ryffel, Bernhard; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB
Subjects: CITOCINAS, DIABETES MELLITUS, DNA MITOCONDRIAL
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ABNT
CARLOS, Daniela et al. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00164. Acesso em: 28 abr. 2024.
APA
Carlos, D., Costa, F. R. C., Pereira, C. A., Rocha, F. A., Yaochite, J. N. U., Oliveira, G. G., et al. (2017). Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.00164
NLM
Carlos D, Costa FRC, Pereira CA, Rocha FA, Yaochite JNU, Oliveira GG, Carneiro FS, Tostes R de CA, Ramos SG, Zamboni DS, Câmara NOS, Ryffel B, Silva JS da. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 abr. 28 ] Available from: https://doi.org/10.3389/fimmu.2017.00164
Vancouver
Carlos D, Costa FRC, Pereira CA, Rocha FA, Yaochite JNU, Oliveira GG, Carneiro FS, Tostes R de CA, Ramos SG, Zamboni DS, Câmara NOS, Ryffel B, Silva JS da. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 abr. 28 ] Available from: https://doi.org/10.3389/fimmu.2017.00164
Artigo de periodico
Host kinin B1 receptor plays a protective role against melanoma progression (2016)
Maria, Andrea G.; Dillenburg-Pilla, Patricia; Reis, Rosana I.; Floriano, Elaine M.; Tefé-Silva, Cristiane; Ramos, Simone Gusmão; Pesquero, João Bosco; Nahmias, Clara; Costa Neto, Cláudio Miguel
Source: Scientific Reports. Unidade: FMRP
Subjects: MELANOMA, CININAS, CALICREINA, PROGNÓSTICO
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ABNT
MARIA, Andrea G. et al. Host kinin B1 receptor plays a protective role against melanoma progression. Scientific Reports, v. 6, 2016Tradução . . Disponível em: https://doi.org/10.1038/srep22078. Acesso em: 28 abr. 2024.
APA
Maria, A. G., Dillenburg-Pilla, P., Reis, R. I., Floriano, E. M., Tefé-Silva, C., Ramos, S. G., et al. (2016). Host kinin B1 receptor plays a protective role against melanoma progression. Scientific Reports, 6. doi:10.1038/srep22078
NLM
Maria AG, Dillenburg-Pilla P, Reis RI, Floriano EM, Tefé-Silva C, Ramos SG, Pesquero JB, Nahmias C, Costa Neto CM. Host kinin B1 receptor plays a protective role against melanoma progression [Internet]. Scientific Reports. 2016 ; 6[citado 2024 abr. 28 ] Available from: https://doi.org/10.1038/srep22078
Vancouver
Maria AG, Dillenburg-Pilla P, Reis RI, Floriano EM, Tefé-Silva C, Ramos SG, Pesquero JB, Nahmias C, Costa Neto CM. Host kinin B1 receptor plays a protective role against melanoma progression [Internet]. Scientific Reports. 2016 ; 6[citado 2024 abr. 28 ] Available from: https://doi.org/10.1038/srep22078
Artigo de periodico
Protective effect of Galectin-1 during Histoplasma capsulatum infection is associated with prostaglandin E2 and nitric oxide modulation (2016)
Rodrigues, Lilian Cataldi; Secatto, Adriana; Sorgi, Carlos Arterio; Dejani, Naiara N.; Medeiros, Alexandra I.; Prado, Morgana Kelly Borges; Ramos, Simone Gusmão; Cummings, Richard D.; Stowell, Sean R.; Faccioli, Lucia Helena; Dias-Baruffi, Marcelo
Source: Mediators of Inflammation. Unidades: FCFRP, FMRP
Subjects: CITOCINAS, HISTOPLASMA, PULMÃO, ÓXIDO NÍTRICO, CITOMETRIA DE FLUXO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, Lilian Cataldi et al. Protective effect of Galectin-1 during Histoplasma capsulatum infection is associated with prostaglandin E2 and nitric oxide modulation. Mediators of Inflammation, v. 2016, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/5813794. Acesso em: 28 abr. 2024.
APA
Rodrigues, L. C., Secatto, A., Sorgi, C. A., Dejani, N. N., Medeiros, A. I., Prado, M. K. B., et al. (2016). Protective effect of Galectin-1 during Histoplasma capsulatum infection is associated with prostaglandin E2 and nitric oxide modulation. Mediators of Inflammation, 2016. doi:10.1155/2016/5813794
NLM
Rodrigues LC, Secatto A, Sorgi CA, Dejani NN, Medeiros AI, Prado MKB, Ramos SG, Cummings RD, Stowell SR, Faccioli LH, Dias-Baruffi M. Protective effect of Galectin-1 during Histoplasma capsulatum infection is associated with prostaglandin E2 and nitric oxide modulation [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 abr. 28 ] Available from: https://doi.org/10.1155/2016/5813794
Vancouver
Rodrigues LC, Secatto A, Sorgi CA, Dejani NN, Medeiros AI, Prado MKB, Ramos SG, Cummings RD, Stowell SR, Faccioli LH, Dias-Baruffi M. Protective effect of Galectin-1 during Histoplasma capsulatum infection is associated with prostaglandin E2 and nitric oxide modulation [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 abr. 28 ] Available from: https://doi.org/10.1155/2016/5813794
Artigo de periodico
Activation of both the calpain and ubiquitin-proteasome systems contributes to septic cardiomyopathy through dystrophin loss/disruption and mTOR inhibition (2016)
Freitas, Ana Carolina Silva; Figueiredo, Maria José; Campos, Erica Carolina; Soave, Danilo Figueiredo; Ramos, Simone Gusmão; Tanowitz, Herbert B.; Celes, Mara Rúbia Nunes
Source: PLOS ONE. Unidade: FMRP
Subjects: MIOCARDIOPATIAS, MODELOS ANIMAIS DE DOENÇAS, EXPRESSÃO GÊNICA, SEPSE, MICROSCOPIA DE FLUORESCÊNCIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FREITAS, Ana Carolina Silva et al. Activation of both the calpain and ubiquitin-proteasome systems contributes to septic cardiomyopathy through dystrophin loss/disruption and mTOR inhibition. PLOS ONE, v. 11, n. 11, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0166839. Acesso em: 28 abr. 2024.
APA
Freitas, A. C. S., Figueiredo, M. J., Campos, E. C., Soave, D. F., Ramos, S. G., Tanowitz, H. B., & Celes, M. R. N. (2016). Activation of both the calpain and ubiquitin-proteasome systems contributes to septic cardiomyopathy through dystrophin loss/disruption and mTOR inhibition. PLOS ONE, 11( 11). doi:10.1371/journal.pone.0166839
NLM
Freitas ACS, Figueiredo MJ, Campos EC, Soave DF, Ramos SG, Tanowitz HB, Celes MRN. Activation of both the calpain and ubiquitin-proteasome systems contributes to septic cardiomyopathy through dystrophin loss/disruption and mTOR inhibition [Internet]. PLOS ONE. 2016 ; 11( 11):[citado 2024 abr. 28 ] Available from: https://doi.org/10.1371/journal.pone.0166839
Vancouver
Freitas ACS, Figueiredo MJ, Campos EC, Soave DF, Ramos SG, Tanowitz HB, Celes MRN. Activation of both the calpain and ubiquitin-proteasome systems contributes to septic cardiomyopathy through dystrophin loss/disruption and mTOR inhibition [Internet]. PLOS ONE. 2016 ; 11( 11):[citado 2024 abr. 28 ] Available from: https://doi.org/10.1371/journal.pone.0166839
Dissertação (Mestrado)
Análise das alterações na musculatura duodenal e resposta do hospedeiro contra infecção pelo Strongyloides venezuelensis e tratamento com Dexametasona: o papel da via JAK-STAT 6 (2016)
Yodono, Nathalia Butschkau Palazzin; Ramos, Simone Gusmão (Orientador)
Unidade: FMRP
Subjects: NEMATOIDES, MÚSCULO LISO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
YODONO, Nathalia Butschkau Palazzin. Análise das alterações na musculatura duodenal e resposta do hospedeiro contra infecção pelo Strongyloides venezuelensis e tratamento com Dexametasona: o papel da via JAK-STAT 6. 2016. Dissertação (Mestrado) – Universidade de São Paulo, Ribeirão Preto, 2016. Disponível em: http://www.teses.usp.br/teses/disponiveis/17/17143/tde-06012017-102934/. Acesso em: 28 abr. 2024.
APA
Yodono, N. B. P. (2016). Análise das alterações na musculatura duodenal e resposta do hospedeiro contra infecção pelo Strongyloides venezuelensis e tratamento com Dexametasona: o papel da via JAK-STAT 6 (Dissertação (Mestrado). Universidade de São Paulo, Ribeirão Preto. Recuperado de http://www.teses.usp.br/teses/disponiveis/17/17143/tde-06012017-102934/
NLM
Yodono NBP. Análise das alterações na musculatura duodenal e resposta do hospedeiro contra infecção pelo Strongyloides venezuelensis e tratamento com Dexametasona: o papel da via JAK-STAT 6 [Internet]. 2016 ;[citado 2024 abr. 28 ] Available from: http://www.teses.usp.br/teses/disponiveis/17/17143/tde-06012017-102934/
Vancouver
Yodono NBP. Análise das alterações na musculatura duodenal e resposta do hospedeiro contra infecção pelo Strongyloides venezuelensis e tratamento com Dexametasona: o papel da via JAK-STAT 6 [Internet]. 2016 ;[citado 2024 abr. 28 ] Available from: http://www.teses.usp.br/teses/disponiveis/17/17143/tde-06012017-102934/
Artigo de periodico
Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset (2016)
Costa, Frederico R. C.; Françozo, Marcela C. S.; Oliveira, Gabriela G. de; Ignacio, Aline; Castoldi, Angela; Zamboni, Dario Simões; Ramos, Simone Gusmão; Câmara, Niels Olsen Saraiva ; Zoete, Marcel R. de; Palm, Noah W.; Flavell, Richard A.; Silva, João Santana da; Carlos, Daniela
Source: Journal of Experimental Medicine. Unidades: FMRP, ICB
Subjects: DIABETES MELLITUS, INTESTINOS, BACTÉRIAS, PROTEÍNAS, DOENÇAS AUTOIMUNES, SISTEMA IMUNE, PÂNCREAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Frederico R. C. et al. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset. Journal of Experimental Medicine, v. 213, n. 7, p. 1223-1239, 2016Tradução . . Disponível em: https://doi.org/10.1084/jem.20150744. Acesso em: 28 abr. 2024.
APA
Costa, F. R. C., Françozo, M. C. S., Oliveira, G. G. de, Ignacio, A., Castoldi, A., Zamboni, D. S., et al. (2016). Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset. Journal of Experimental Medicine, 213( 7), 1223-1239. doi:10.1084/jem.20150744
NLM
Costa FRC, Françozo MCS, Oliveira GG de, Ignacio A, Castoldi A, Zamboni DS, Ramos SG, Câmara NOS, Zoete MR de, Palm NW, Flavell RA, Silva JS da, Carlos D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset [Internet]. Journal of Experimental Medicine. 2016 ; 213( 7): 1223-1239.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1084/jem.20150744
Vancouver
Costa FRC, Françozo MCS, Oliveira GG de, Ignacio A, Castoldi A, Zamboni DS, Ramos SG, Câmara NOS, Zoete MR de, Palm NW, Flavell RA, Silva JS da, Carlos D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset [Internet]. Journal of Experimental Medicine. 2016 ; 213( 7): 1223-1239.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1084/jem.20150744
Artigo de periodico
Reduced expression of adherens and gap junction proteins can have a fundamental role in the developmentof heart failure following cardiac hypertrophy in rats (2016)
Santos, Daniele O. dos; Blefari, Valdecir; Prado, Fernanda P.; Silva, Carlos A.; Fazan Júnior, Rubens; Salgado, Hélio César; Ramos, Simone Gusmão; Prado, Cibele Maria
Source: Experimental and Molecular Pathology. Unidade: FMRP
Subjects: HIPERTENSÃO, PROTEÍNAS, INSUFICIÊNCIA CARDÍACA, HIPERTROFIA VENTRICULAR ESQUERDA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Daniele O. dos et al. Reduced expression of adherens and gap junction proteins can have a fundamental role in the developmentof heart failure following cardiac hypertrophy in rats. Experimental and Molecular Pathology, v. 100, n. 1, p. 167- 176, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.yexmp.2015.12.009. Acesso em: 28 abr. 2024.
APA
Santos, D. O. dos, Blefari, V., Prado, F. P., Silva, C. A., Fazan Júnior, R., Salgado, H. C., et al. (2016). Reduced expression of adherens and gap junction proteins can have a fundamental role in the developmentof heart failure following cardiac hypertrophy in rats. Experimental and Molecular Pathology, 100( 1), 167- 176. doi:10.1016/j.yexmp.2015.12.009
NLM
Santos DO dos, Blefari V, Prado FP, Silva CA, Fazan Júnior R, Salgado HC, Ramos SG, Prado CM. Reduced expression of adherens and gap junction proteins can have a fundamental role in the developmentof heart failure following cardiac hypertrophy in rats [Internet]. Experimental and Molecular Pathology. 2016 ; 100( 1): 167- 176.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.yexmp.2015.12.009
Vancouver
Santos DO dos, Blefari V, Prado FP, Silva CA, Fazan Júnior R, Salgado HC, Ramos SG, Prado CM. Reduced expression of adherens and gap junction proteins can have a fundamental role in the developmentof heart failure following cardiac hypertrophy in rats [Internet]. Experimental and Molecular Pathology. 2016 ; 100( 1): 167- 176.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.yexmp.2015.12.009
Artigo de periodico
5-lipoxygenase pathway is essential for the control of granuloma extension induced by Schistosoma mansoni eggs in lung (2016)
Silva, Gabriel Toffoli da; Espíndola, Milena Sobral; Fontanari, Carolina; Rosada, Rogerio Silva; Faccioli, Lucia Helena; Ramos, Simone Gusmão; Rodrigues, Vanderlei; Frantz, Fabiani Gai
Source: Experimental Parasitology. Unidades: FCFRP, FMRP
Subjects: LIPOXIGENASE, SCHISTOSOMA MANSONI, GRANULOMA, ESQUISTOSSOMOSE, LIPÍDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Gabriel Toffoli da et al. 5-lipoxygenase pathway is essential for the control of granuloma extension induced by Schistosoma mansoni eggs in lung. Experimental Parasitology, v. 167, p. 124-129, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.exppara.2016.06.001. Acesso em: 28 abr. 2024.
APA
Silva, G. T. da, Espíndola, M. S., Fontanari, C., Rosada, R. S., Faccioli, L. H., Ramos, S. G., et al. (2016). 5-lipoxygenase pathway is essential for the control of granuloma extension induced by Schistosoma mansoni eggs in lung. Experimental Parasitology, 167, 124-129. doi:10.1016/j.exppara.2016.06.001
NLM
Silva GT da, Espíndola MS, Fontanari C, Rosada RS, Faccioli LH, Ramos SG, Rodrigues V, Frantz FG. 5-lipoxygenase pathway is essential for the control of granuloma extension induced by Schistosoma mansoni eggs in lung [Internet]. Experimental Parasitology. 2016 ; 167 124-129.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.exppara.2016.06.001
Vancouver
Silva GT da, Espíndola MS, Fontanari C, Rosada RS, Faccioli LH, Ramos SG, Rodrigues V, Frantz FG. 5-lipoxygenase pathway is essential for the control of granuloma extension induced by Schistosoma mansoni eggs in lung [Internet]. Experimental Parasitology. 2016 ; 167 124-129.[citado 2024 abr. 28 ] Available from: https://doi.org/10.1016/j.exppara.2016.06.001
Artigo de periodico
Effects of methylene blue in acute lung injury induced by oleic acid in rats (2016)
Silveira, Ana Paula Cassiano; Vento, Daniella Alves; Albuquerque, Agnes Afrodite Sumarelli; Celotto, Andrea Carla; Tefé-Silva, Cristiane; Ramos, Simone Gusmão; Nadai, Tales Rubens de; Rodrigues, Alfredo José; Poli Neto, Omero Benedicto; Evora, Paulo Roberto Barbosa
Source: Annas of Translational Medicine. Unidade: FMRP
Subjects: MODELOS ANIMAIS DE DOENÇAS, ENDOTÉLIO, HOMEOSTASE, DOENÇAS PULMONARES INTERSTICIAIS, EDEMA PULMONAR, PULMÃO, ÁCIDOS GRAXOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVEIRA, Ana Paula Cassiano et al. Effects of methylene blue in acute lung injury induced by oleic acid in rats. Annas of Translational Medicine, v. 4, n. 1, 2016Tradução . . Disponível em: https://doi.org/10.3978/j.issn.2305-5839.2015.12.51. Acesso em: 28 abr. 2024.
APA
Silveira, A. P. C., Vento, D. A., Albuquerque, A. A. S., Celotto, A. C., Tefé-Silva, C., Ramos, S. G., et al. (2016). Effects of methylene blue in acute lung injury induced by oleic acid in rats. Annas of Translational Medicine, 4( 1). doi:10.3978/j.issn.2305-5839.2015.12.51
NLM
Silveira APC, Vento DA, Albuquerque AAS, Celotto AC, Tefé-Silva C, Ramos SG, Nadai TR de, Rodrigues AJ, Poli Neto OB, Evora PRB. Effects of methylene blue in acute lung injury induced by oleic acid in rats [Internet]. Annas of Translational Medicine. 2016 ; 4( 1):[citado 2024 abr. 28 ] Available from: https://doi.org/10.3978/j.issn.2305-5839.2015.12.51
Vancouver
Silveira APC, Vento DA, Albuquerque AAS, Celotto AC, Tefé-Silva C, Ramos SG, Nadai TR de, Rodrigues AJ, Poli Neto OB, Evora PRB. Effects of methylene blue in acute lung injury induced by oleic acid in rats [Internet]. Annas of Translational Medicine. 2016 ; 4( 1):[citado 2024 abr. 28 ] Available from: https://doi.org/10.3978/j.issn.2305-5839.2015.12.51

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